I can't find the link offhand, but a new study was publised that there is a significant increase in the number of satellite cells with IGF-1 co-localization after the workout. This just kind of corroborates what we already know, and adds more evidence against the fallacy of only looking at protein synthesis rates when it's one piece of the puzzle. Also, helps provide even more evidence against trying to increase systemic GH/IGF-1 when in fact it's the autocrine/paracrine produced somatomedins that are important.
Quote (Lightman @ Dec 23 2013 04:36am)
The study, involving Harvard University and the University of NSW, discovered a way of restoring the efficiency of cells, completely reversing the ageing process in muscles.
Could this refer to subsequent atrophy, rediffusion of ATP-CP decompounding moles, loss of strength and sarcoplasmic cross sectional width?
Because technically, hypertrophy is damaged tissue. Restoring said mitochondrial connection would speculatively also mitigate the needed overflow in energy processes & citric cycling for hypertrophized cells.
or it could mean the exact opposite, and open a new frontier in neural stress adaptations.
m9 I have no idea
I guess I just found it interesting that from the article they are only injecting NAD+. I just wouldn't expect those kinds of effects from only that, knowing its role in the krebs cycle