"Dose Response of 1, 3 and 5 Sets of Resistance Exercise on Strength, Local Muscular Endurance and Hypertrophy."

http://journals.lww.com/nsca-jscr/Abstract/publishahead/Dose_Response_of_1,_3_and_5_Sets_of_Resistance.97085.aspx

"1 maximal set == 3,5 sets"
hehehe

https://www.facebook.com/permalink.php?story_fbid=325977817608705&id=175556779317477

RM1 charts, tailor your own - stop following a lie.

"Diurnal rhythms of plasma GLP-1 levels in normal and overweight/obese subjects: lack of effect of weight loss."

http://www.ncbi.nlm.nih.gov/pubmed/25543251

uh oh

In addition to the RM topic-
Person A

Person B

Person C


^how E prediction (blue line) falls in line near perfectly with reality (orange line) whereas regular 2.5% predictors just don't.

Wot
http://www.ncbi.nlm.nih.gov/m/pubmed/10838463/
Somebody check this study out pls

casein protein superior?

Nah not at all
but i can't read full text.. you'd probably have to factor in a self-reported intake for the total kcals consumed and that's likely where the problem arises

"Quantitative Feed Restriction Rather Than Caloric Restriction Modulates the Immune Response of Growing Rabbits"

http://jn.nutrition.org/content/early/2015/01/07/jn.114.197871.abstract

can't find full text but it may indicate more corroboration for an anti-inflammatory effect of intermittent fasting

"Activation of the hypothalamic-pituitary-adrenal stress axis induces cellular oxidative stress"

http://journal.frontiersin.org/Journal/10.3389/fnins.2014.00456/abstract

You can find the full text for this by googling the title. Pretty interesting though.
This actually brings up something I knew very little about. The ability of cortisol and thus stress to directly and indirectly upregulate ROS production. Very interesting and informative.

"Comparison with ancestral diets suggests dense acellular carbohydrates promote an inflammatory microbiota, and may be the primary dietary cause of leptin resistance and obesity"

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3402009/pdf/dmso-5-175.pdf

Something I've been talking about for a little bit now, though it's overall still a bit nebulous. The ability of certain diets to increase intestinal permeability and LPS absorption via modulating the gut microbiome, thus culminating in a chronical elevation of leptin and leptin resistance in a prolonged state. That could easily lead to obesity.. it could easily be that something like this could precipitate obesity. Although fat loss is absolutely a thermodynamic phenomenon, if you ate the prototypical "shitty" diet almost completely, it's possible you're making it much easier on yourself to become obese via these gut changes through dysfunctional satiety signals and such.

"Resistance Training Recovery: Considerations for Single vs. Multi-joint Movements and Upper vs. Lower Body Muscles"

http://digitalcommons.wku.edu/ijes/vol8/iss1/10/

"β-Aminoisobutyric Acid Induces Browning of White Fat and Hepatic β-oxidation and is Inversely Correlated with Cardiometabolic Risk Factors"

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4017355/

Ah this is great. Further elucidation into the myokine response to exercise.. this particularly activates PGC-1 alpha. I'll need to revise my paradigm of how white fat induction into beige fat likely works via exercise and cold thermogenesis now. Just another player to add, though it likely fits in an early step, facilitating the robust PGC-1 alpha response early on. Very cool.
It also seems this substance may begin being sold as a supplement..

"Mutagenicity and DNA-damaging potential of clenbuterol and its metabolite 4-amino-3,5-dichlorobenzoic acid in vitro."

http://www.ncbi.nlm.nih.gov/pubmed/25595371

So.. not super harmful

"Comparison of muscle hypertrophy following 6-month of continuous and periodic strength training"

http://link.springer.com/article/10.1007/s00421-012-2511-9

Cool as fuck. It essentially shows that cycling between 6 weeks of lifting, then 3 weeks of deloading and repeating that sequence for 24 weeks produces virtually equal results to training straight for 24 weeks.

study discusses its many flaws in discussion such as inaccuracy of measurement... there probably was no blinding and unbiased measurement as well (performance + detection bias) - synergistically BAD with the inaccuracy.

(also the fact that across all 3 groups, there weren't even 40 people and there was a massive attrition and so on)

if the ''bottom half" of all research were to disappear tomorrow, the world might be a better place....

inafter - articles people wrote just trying to hustle for that M.Sc degree