no
as in the lactace and metabolites I talked about

How would it result in no difference in hypertrophy in that study on amount of reps to promote hypertrophy u were talking about?

It's just surmise
I haven't really delved into that study fully and really thought about it. However, from a cursory understanding, that seems likely. Check the other variables I talked of and think about the differences that would stem from different training types, as well as ways to mitigate that

I feel like that idea of hypertrophy is working the muscle until it like explodes though

But let's be honest, does the body count your reps? Can it tell? Or is it just the right amount of work the muscle does? And the strength gains connected with lower reps is more due to neural adaptation and the less mass associated with it is because you aren't working the muscle as much (small amount of sets for low reps 3x5 compared to hypertrophys 5 sets of 12.)

http://www.zentrum-der-gesundheit.de/pdf/intermittierendes-fasten-ia-03.pdf

Text on if approach

Just a simple question;

I've always heard that your body cannot burn trans fatty acids and that they stay in your body forever. I haven ever pursued any sort of research or proof of this claim.

Can any of you shed some light on the truth of this subject?

Thanks

your body simply responds to absolute volumes of work. the more work you do the greater the net flux through metabolic pathways and ultimately the greater the load on the cellular environment. the further you push your body from homeostasis (within reason) the more likely you are to challenge adaptive mechanisms such as hypertrophy, enzymatic production, energy stores etc

high resistance, low volume scenarios are not supreme challenges to metabolic homeostasis imo.



http://ajcn.nutrition.org/content/34/10/2307.full.pdf+html

references several studies that trans isomers are oxidized at similar or slightly slower rates than cis isomers of fatty acids, so no what you heard is not true but may stem from evidence suggesting that trans isomers may linger in tissue lipids slightly longer than cis isomers. essentially the body has methods for dealing with dietary trans isomers of fatty acids, and evidence suggests that in proper ratios dietary transfats are not harmful

This post was edited by cloudkicker on Apr 22 2014 04:07pm

"Enhancement of Memories by Systemic Administration of Insulin-Like Growth Factor II"

http://www.nature.com/npp/journal/vaop/ncurrent/abs/npp201469a.html

This is obviously clear, as mTOR influence on long term memory (late phase LTP, where protein synthesis is required) is obvious. Still pretty sweet however. Brb injecting IGF into my hippocampus.. no possible ramifications there

Impact of diet on adult hippocampal neurogenesis

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2775886/

more benefits of fasting:

"We have also found that independent of calorie intake, meal frequency is a key player in modulating AHN. Indeed, without reducing calorie intake, extending the time between meals increases AHN. It also changes hippocampal gene expression and correlates with performance in hippocampus-dependent tasks and mood".

Even more fasting benefits for cognitive function. Stacking up a nice list, even down to changes in gene expression and neuroprotection.

http://www.ncbi.nlm.nih.gov/pubmed/23608698

http://i.imgur.com/azh7W1I.jpg?1