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Aug 30 2015 10:11pm
Quote (Qwaze @ Aug 31 2015 03:33am)
gotta 2nd this

also casein can take up to 4-7 hours to digest because it clots in your stomach, its ideal before bed or before a period where you expect to fast a couple hours (keeps the hunger away and slow release of amino acids in the blood), casein is extracted from milk during the cheese making process, so its a balanced and complete profile of AAs

TLDR: Casein before a fast/before bed, whey/soy or whatever any other time

Casein is usually more expensive though, thats another thing to keep in mind, but it definitely has its uses

e/ lol thread is pretty much 6inch turkey ego boost



I wouldn't eat soy protein cuz that shit be estrogen
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Aug 30 2015 11:39pm
Quote (dark-soul @ Aug 30 2015 11:33pm)
That's what the professor who's an rd taught my nutrition class a few semesters ago. Here's one source backing that although I spent 2 secs on google.

http://www.heart.org/HEARTORG/GettingHealthy/NutritionCenter/HealthyEating/Saturated-Fats_UCM_301110_Article.jsp


Your rd was not up on the science then, just recapitulating dogma from previous years

http://ajcn.nutrition.org/content/91/3/535
https://www.ncbi.nlm.nih.gov/pubmed/26268692
http://annals.org/article.aspx?articleid=1846638
http://archinte.jamanetwork.com/article.aspx?articleid=1108492
http://www.karger.com/Article/Pdf/229002

Overall, it's not as bad as previously thought because it commensurately raises both HDL & LDL, thereby not increasing the risk too much. What I can concede, though, is that excessive intakes of SFAs will favor LDL accretion. There may be some benefit of replacing excessive intakes w/ PUFAs or MUFAs though it also probably depends on genetic and lifestyle factors.
There's also a wealth of scrutinization denoting that SFA intake also doesn't correlate well with insulin resistance, as is often pontificated. It's interesting too, as the deleterious effects of inflammation are well known in the pathology of both insulin resistance and CVD, and SFAs have a couple of different mechanisms delineated to cause an inflammatory response, namely augmenting LPS infiltration through the brush border and directly activating TLR4 signaling, both of which should lead to ill effects and more robust correlations in these ailments. However, this leads me back to the genetic & lifestyle thing - probably explains why some will find a benefit and others (many others in fact) will find little to none to SFA restriction/replacement. The quality may also matter tbh. So perhaps would the cooking method and temperature used.
The point being - the debate on SFA intake is still open - current evidence and thought points to it actually not being harmful, though notwithstanding, there is still a good deal of evidence pointing the other way as well. My personal thought is that it's largely benign.
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Aug 31 2015 12:00am
Quote (Balla @ Aug 30 2015 11:39pm)
Your rd was not up on the science then, just recapitulating dogma from previous years

http://ajcn.nutrition.org/content/91/3/535
https://www.ncbi.nlm.nih.gov/pubmed/26268692
http://annals.org/article.aspx?articleid=1846638
http://archinte.jamanetwork.com/article.aspx?articleid=1108492
http://www.karger.com/Article/Pdf/229002

Overall, it's not as bad as previously thought because it commensurately raises both HDL & LDL, thereby not increasing the risk too much. What I can concede, though, is that excessive intakes of SFAs will favor LDL accretion. There may be some benefit of replacing excessive intakes w/ PUFAs or MUFAs though it also probably depends on genetic and lifestyle factors.
There's also a wealth of scrutinization denoting that SFA intake also doesn't correlate well with insulin resistance, as is often pontificated. It's interesting too, as the deleterious effects of inflammation are well known in the pathology of both insulin resistance and CVD, and SFAs have a couple of different mechanisms delineated to cause an inflammatory response, namely augmenting LPS infiltration through the brush border and directly activating TLR4 signaling, both of which should lead to ill effects and more robust correlations in these ailments. However, this leads me back to the genetic & lifestyle thing - probably explains why some will find a benefit and others (many others in fact) will find little to none to SFA restriction/replacement. The quality may also matter tbh. So perhaps would the cooking method and temperature used.
The point being - the debate on SFA intake is still open - current evidence and thought points to it actually not being harmful, though notwithstanding, there is still a good deal of evidence pointing the other way as well. My personal thought is that it's largely benign.


Thank you for the response. I wasn't aware it raised HDLs as well which would most likely reduce the risk since HDL/LDL ratio seems to be the main concern.
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Aug 31 2015 05:23am
Quote (6inchturkey @ Aug 30 2015 11:54pm)
Yooo dats pro fucking source brooo, wooooot his teacher is demigod of knowledge. I actually google all subjects and "extensively" read all sides and then come up with my own answer and i try it. Taken sat fats mainly coconut.oil and ground beef for years, im healthy as fuck. Ofc moderation is key


this just made u sound stupid

u google stuff, believe what you want, try it out, and self diagnose yourself as healthy

cause u do something and u think you are healthy means it works for everyone, and works for u, with nothing the backup the claim, genius

if only every1 did this...
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Aug 31 2015 06:58am
Quote (Balla @ Aug 31 2015 01:39am)
Your rd was not up on the science then, just recapitulating dogma from previous years

http://ajcn.nutrition.org/content/91/3/535
https://www.ncbi.nlm.nih.gov/pubmed/26268692
http://annals.org/article.aspx?articleid=1846638
http://archinte.jamanetwork.com/article.aspx?articleid=1108492
http://www.karger.com/Article/Pdf/229002

Overall, it's not as bad as previously thought because it commensurately raises both HDL & LDL, thereby not increasing the risk too much. What I can concede, though, is that excessive intakes of SFAs will favor LDL accretion. There may be some benefit of replacing excessive intakes w/ PUFAs or MUFAs though it also probably depends on genetic and lifestyle factors.
There's also a wealth of scrutinization denoting that SFA intake also doesn't correlate well with insulin resistance, as is often pontificated. It's interesting too, as the deleterious effects of inflammation are well known in the pathology of both insulin resistance and CVD, and SFAs have a couple of different mechanisms delineated to cause an inflammatory response, namely augmenting LPS infiltration through the brush border and directly activating TLR4 signaling, both of which should lead to ill effects and more robust correlations in these ailments. However, this leads me back to the genetic & lifestyle thing - probably explains why some will find a benefit and others (many others in fact) will find little to none to SFA restriction/replacement. The quality may also matter tbh. So perhaps would the cooking method and temperature used.
The point being - the debate on SFA intake is still open - current evidence and thought points to it actually not being harmful, though notwithstanding, there is still a good deal of evidence pointing the other way as well. My personal thought is that it's largely benign.


cardiovascular exercise is a great clearance mechanism for SF's, reduces LDL in blood as SF's are actually used for training instead of just shuttling around in the blood until they adhere to an artery wall. my beef with current medicine is that they need exercise conditions as well as control and intervention conditions for each experiment, rather than just using sedentary fucks to begin with. sedentary populations should not be the only standard we hold ourselves to in research.
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Aug 31 2015 12:35pm
Quote (cloudkicker @ Aug 31 2015 08:58am)
cardiovascular exercise is a great clearance mechanism for SF's, reduces LDL in blood as SF's are actually used for training instead of just shuttling around in the blood until they adhere to an artery wall. my beef with current medicine is that they need exercise conditions as well as control and intervention conditions for each experiment, rather than just using sedentary fucks to begin with. sedentary populations should not be the only standard we hold ourselves to in research.


i see what u did there

beef :D
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Aug 31 2015 01:35pm
This post is a violation of the site rules and appropriate action was taken.

Quote (noob_whacker @ Aug 31 2015 11:23am)
this just made u sound stupid

u google stuff, believe what you want, try it out, and self diagnose yourself as healthy

cause u do something and u think you are healthy means it works for everyone, and works for u, with nothing the backup the claim, genius

if only every1 did this...


coming from the retard. hahah such irony
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Aug 31 2015 02:10pm
Quote (6inchturkey @ Aug 31 2015 03:35pm)
coming from the retard. hahah such irony


are you wretch multi?

u sound like a 5 year old... im done
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Aug 31 2015 06:59pm
This post is a violation of the site rules and appropriate action was taken.

Quote (noob_whacker @ Aug 31 2015 08:10pm)
are you wretch multi?

u sound like a 5 year old... im done


the retard strikes again.....
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