Hiho,

This isn't a homework exercise, but more of a loose thought I hope somebody could debunk for me. I couldn't find it out myself.
The lagging strand of DNA (during the transcription process) is read in small pieces with new RNA primers being necessary to read the next piece of the strand. These small pieces each form an okazaki fragments.
Do these fragments correlate in any way to the amount of introns that are copied from the strand? I can imagine that if the transcription stops at one point, there shouldn't be an RNA primer until after the next intron, thus decreasing the amount of introns in RNA formed from lagging strands. This would increase the speed of the modification of mRNA and thus the translation process.

This doesn't make any sense, the lagging strands is called just that for a reason.... ..Right?

Post-translational modification occurs after DNA has been converted into RNA. The primers you are referring to are not selective in the method that you have described.

So then how are introns of the lagging strand dealed with?

Are you referring to DNA replication? There is no lagging strand in transcription, unless I'm mistaken...